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Nasal Deformities

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A congenital or traumatically acquired bending or bowing of the nasal septum The modern surgical treatment of chronic sinusitis is performed intranasally under ... – PowerPoint PPT presentation

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Title: Nasal Deformities


1
Nasal Deformities
Septal Deviation
A congenital or traumatically acquired
bending or bowing of the nasal septum
2
Symptoms
  • Mild forms do not cause
  • symptoms and have no pathologic significance

3
Symptoms
  • More pronounced degrees of septal curvature can
    obstruct nasal breathing and may also cause
    olfactory impairment due to inadequate
    ventilation of the olfactory groove.
  • Deficient nasal airflow can also lead to
    paranasal sinus sequelae such as headaches and
    recurrent sinusitis.
  • A large septal spur that comes into
  • contact with the nasal turbinates can
    cause epistaxis

4
Diagnosis
  • Septal subluxation is a special form in which the
    anterior septal margin is displaced from the
    median plane. This condition is readily
    identified by external inspection of the nasal
    base.
  • Further clinical examination consists of anterior
    rhinoscopy or endoscopy.
  • The degree of nasal obstruction can be
    objectively evaluated by rhinomanometry.
  • For medicolegal reasons, olfactory testing should
    always be done prior to surgical treatment

5
Treatment
  • The treatment of choice is surgical straightening
    of the deviated septum (septoplasty)

6
Deformities of the External Nose
Causes and forms
Deformities may be congenital or traumatically
acquired The most common deformities are a
crooked nose, humped nose, saddle nose, and broad
nose, which may occur separately or in
combinations
7
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8
Diagnosis
  • Inspection
  • Anterior rhinoscopy
  • Endoscopy

9
Treatment
  • The treatment of choice is functional
    septorhinoplasty, with correction of the nasal
    septum and external nose

10
Nosebleed (Epistaxis)
  • Nosebleed is a relatively common, usually
    harmless symptom that may reflect a number of
    diseases of variable severity

11
Local causes
  • Perforation
  • traumatic
  • iatrogenic
  • Inflammatory
  • spurs or ridges
  • Foreign bodies
  • rhinoliths
  • trauma (including nose picking)
  • allergy
  • acute rhinitis
  • Traumatic aneurysm of the internal carotid
  • Benign neoplasms
  • malignant neoplasms

12
Systemic disease
  • Atherosclerosis
  • Infection
  • Pregnancy
  • Diabetes mellitus
  • Congenital e.g., hemophilia A and B, Willebrand
    disease
  • Acquired e.g., anticoagulant therapy,
  • Hepatocellular insufficiency
  • Platelet disorders
  • Congenital
  • Acquired uremia, dysproteinemia, adverse effects
    of dextrann and acetylsalicylic acid (ASA)
    therapy SchönleinHenoch purpura
  • Osler disease

13
Diagnosis
  • Nosebleed requires a simultaneous, coordinated
    protocol of diagnostic and therapeutic actions

14
Evaluation
  • The diagnostic work-up begins with blood pressure
    measurement.
  • Except in very minor cases, the Hb should also be
    determined, and a coagulation disorder should be
    excluded by determining the platelet count,
    bleeding time, thromboplastin time, partial
    thromboplastin time (PTT), and thrombin time

15
Bleeding Site
  • The nasal cavity is inspected by anterior
    rhinoscopy or endoscopy following decongestion
    and local anesthesia of the mucosa.
  • In most cases the bleeding site is in
    Kiesselbachs area

16
Treatment
  • General measures
  • The nostrils are compressed against the nasal
    septum
  • the patient is told not to swallow blood running
    down the pharynx.
  • The patient is kept in an upright posture
  • An ice bag can be placed on the back of the neck
    to induce reflex vasoconstriction
  • An intravenous line should be placed if bleeding
    is severe

17
Silver nitrate cautery
  • Mild epistaxis from Kiesselbachs area can often
    be controlled by selective local cauterization

18
Nasal packing
  • For severe epistaxis, the anterior nasal cavity
    can be packed with ointment-impregnated gauze
    strips

19
Vascular ligation or embolization
  • The most common source of bleeding from the
    posterolateral part of the nasal cavity is the
    sphenopalatine artery (branch of the maxillary
    artery), which can be coagulated or clipped under
    endoscopic control

20
Surgical prevention of recurrent epistaxis
  • The main indications for surgery are changes in
    the nasal septum such as septal spurs, ridges,
    and perforations.
  • Treatment consists of straightening the nasal
    septum (septoplasty or closing the septal
    perforation (e.g., by implanting an auricular
    cartilage graft and using local mucosal flap
    advancement

21
Fractures of the Nasal Pyramid and Lateral Midface
22
Nasal Pyramid Fracture
  • The nasal pyramid is predisposed to fractures
    because of its exposed location.

23
Diagnostic procedure
  • Inspection

24
Palpation
  • Crepitus noted on palpation confirms the
    suspicion of a fracture

25
Imaging
  • Further diagnostic measures include radiographs
    of the nose in the lateral projection
  • Standard sinus projections to exclude bony
    involvement of the lateral midface

26
Complications
  • Subperichondrial hemorrhage with hematoma
  • Septal Abcess

27
Lateral Midfacial Fractures
  • Lateral midfacial fractures are usually caused by
    blunt trauma to the side of the face.
  • Affected structures of the bony facial skeleton
    are the maxillary sinus, orbit, and the zygoma or
    zygomatic arch

28
  • An isolated fracture of the orbital floor with a
    partial herniation of the orbital contents into
    the maxillary sinus is a special type of lateral
    midfacial fracture called a blow-out fracture

29
Symptoms
  • Facial asymmetry
  • Limited mouth opening
  • Diplopia
  • Sensory disturbances

30
Diagnosis
  • Inspection
  • Swelling
  • subcutaneous hemorrhage
  • Asymmetry of the affected facial
  • Enophthalmos

31
  • Palpation
  • Concomitant soft-tissue swelling can make it
    difficult or impossible to palpate sites of bony
    discontinuity or displacement

32
  • Sensory testing
  • Wisps of cotton can be used to test sensory
    function on the healthy and affected sides

33
  • Radiographs
  • Whenever a lateral midfacial fracture is
    suspected, standard sinus radiographs should be
    obtained (occipitomental and occipitofrontal
    projections to define the extent of the bony
    discontinuity or displacement

34
  • The zygomatic arches may be poorly visualized in
    standard projections, and so a bucket handle
    view should be added when a concomitant zygomatic
    arch fracture is suspected

35
  • CT Scans
  • be helpful to obtain a more discriminating view
    of the fracture and also to exclude an
    involvement of the anterior skull base

36
Inspection
37
Imaging
38
Imaging
39
Treatment
  • Surgical treatment
  • is unnecessary for undisplaced, asymptomatic
    fractures
  • is indicated for displaced fractures or fractures
    that are causing symptoms such as sensory
    deficits in the distribution of the infraorbital
    nerve, diplopia on upward gaze, enophthalmos,
    restricted jaw opening, or facial asymmetry.
  • Treatment consists of reduction and fixation of
    the bone fragments using miniplates, interosseous
    wiring, or both

40
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41
Fractures of the Central Midfaceand Anterior
Skull Base
42
Classification
  • Central midfacial fractures (Le Fort I-III)
  • Frontobasal fractures (Escher classification)

43
Le Fort classification
44
Escher classification
45
Notice
  • Frontobasal fractures occupy a special place
    among skull fractures because they are usually an
    indirectly open injury that creates a
    communication between the cranial cavity and the
    environment lead to life-threatening intracranial
    complications (e.g., meningitis, brain abscess)

46
Symptoms
  • Unilateral or bilateral periorbital hematoma
  • Dish face the midface has been separated from
    the skull base and displaced inward
  • Cerebrospinal fluid (CSF) rhinorrhea
  • Vision loss
  • Diplopia
  • Cerebral prolapse
  • Anosmia

47
Examination
48
CSF Leakage
49
Detection of CSF
  • Glucose test
  • ß2-transferrin

50
Diagnosis
  • Computed tomography
  • Axial scans are for evaluating the anterior and
    posterior walls of the frontal sinuses and
    sphenoid sinus
  • Coronal scans more clearly define the ethmoid
    roof and cribriform plate

51
Complementary tests
  • Testing of hearing and balance
  • Olfactory testing

52
Treatment
  • Every confirmed fracture of the anterior skull
    base should be treated surgically in operable
    patients, regardless of whether or not a CSF leak
    has been detected

53
Indications for surgery
54
Vital indications (operate immediately)
  • Life-threatening rise of intracranial pressure
    due to intracranial hemorrhage
  • Bleeding from the nose or sinuses that is
    refractory to conservative treatment
  • Bleeding from an open skull injury that is
    refractory to conservative treatment

55
Absolute indications (operate as soon as
possible)
  • Open brain injury
  • Dural tear from an indirectly open head injury
  • Penetrating foreign bodies and impalement
    injuries
  • Early complications (e.g., meningitis,
    encephalitis, brain abscess)
  • Late complications (e.g., meningitis, brain
    abscess, osteomyelitis)
  • Orbital complications

56
Relative indications (operate in 12 weeks)
  • Displaced bone fragments
  • Fractures involving the drainage tracts of the
    paranasal sinuses (ostiomeatal unit)
  • Acute or chronic sinusitis at the time of the
    injury
  • Post-traumatic sinus inflammation, mucopyocele
    formation
  • Supraorbital nerve injury due to an adjacent
    fracture

57
Questions
  • Define the paradoxical cyanosis.
  • Name four common nasal deformity.
  • Where is the common site of epistaxis in old age?
  • What is the most definitive sign for nasal
    fracture?
  • Name six common symptoms for frontobasal
    fracture.

58
Session 4
  • Inflammations of the External Nose, Nasal Cavity,
  • and Facial Soft Tissues

59
Inflammations of the Hair Follicles
  • Folliculitis the disease is confined to the hair
    follicles.
  • Furuncle the infection spreads to deeper
    tissues and forms a central core of purulent
    liquefaction.

60
Symptoms
  • Nasal furuncles present as painful, tender,
    erythematous swellings about the nasal tip and
    nares

61
Treatment
  • Antibiotic that is active against staphylococci
  • Dicloxacillin sodium , Cephalexin and so on
  • Combined with the local application of an
    antibiotic-containing ointment

62
Complications
  • Inadequate treatment or manipulations of the
    nasal furuncle itself can result in
  • Hematogenous spread to intracranial
    structures

63
Erysipelas
  • Causative organisms are beta-hemolytic group A
    streptococci
  • Less common pathogens are streptococci of other
    groups, Staphylococcus aureus, and gram-negative
    rods (e.g., Klebsiella pneumoniae)

64
Symptoms
  • High fever
  • Feeling of tension in the soft tissues
  • Rapidly by broad areas of erythema and swelling,
    which are sharply demarcated from unaffected skin
  • The tissue is warm to the touch, and small
    blisters occasionally form

65
Treatment
  • The treatment of choice is the parenteral
    administration of penicillin

66
Inflammations of the Nasal Cavity
67
Acute Rhinitis
  • Acute rhinitis (common cold) is the most
    prevalent infectious disease
  • Rhinoviruses and coronaviruses comprise almost
    half of the causative organisms of acute viral
    rhinitis

68
Symptoms
  • Dry stage
  • Malaise (lethargy, headache, fever) and local
    discomfort in the nose and nasopharynx (burning,
    soreness).
  • Catarrhal stage
  • Watery, initially serous nasal discharge and
    nasal obstruction due to mucosal swelling, which
    mainly involves the turbinates.

69
Bacterial Superinfection
  • Viral damage to the epithelium promotes bacterial
    colonization, which alters the consistency of the
    clear nasal discharge, causing it to become
    mucopurulent.

70
Treatment
  • Treatment consists of supportive measures to
    relieve nasal obstruction and prevent sinusitis
    and other sequelae by the use of decongestant
    nose drops
  • Antibiotics may also be prescribed in patients
    with bacterial superinfection or paranasal sinus
    involvement

71
Nonspecific Chronic Rhinitis
  • Nonspecific chronic rhinitis can develop due to
    anatomic changes (e.g., marked septal deviation,
    septal spur) or other lesions of the nasal cavity
    (polyps, tumors) and nasopharynx (adenoids)
  • Environmental factors such as sustained extreme
    temperatures or air pollutants can also bring on
    this condition

72
Symptoms
  • Patients present clinically with
  • Obstructed nasal breathing
  • Mucous nasal discharge
  • Frequent throat clearing and occasional
    hoarseness

73
Treatment
  • The most important step is to eliminate the cause
    by removing chronic irritants from the
    environment or by surgically correcting any
    intranasal pathology (e.g., septoplasty)

74
Specific Chronic Rhinitis
  • Tuberculosis
  • Sarcoidosis
  • Rhinoscleroma
  • Actinomycosis
  • Syphilis
  • Wegner Granulomatosis
  • Fungal infections
  • Aspergillosis
  • Mucormycosis
  • Rhinosporidiosis

75
Allergic Rhinitis
  • Triggered by an immediate, IgE-mediated reaction
    of the immune system to any of a number of
    foreign substances, particularly pollens and
    animal allergens.

76
Seasonal allergic rhinitis
  • Mainly by pollens
  • Disappear at the end of the pollen season

77
Perennial allergic rhinitis
  • Is caused by year-round allergen exposure
  • The predominant causative allergens are house
    dust, pet dander, and molds
  • The disease may also be caused by certain foods
    (e.g., strawberries, nuts, eggs, fish) as well as
    occupational exposure to allergens (e.g., bakers
    and hairdressers)

78
Symptoms
  • The clinical manifestations
  • Obstructed nasal breathing
  • Sneezing attacks
  • Watery nasal discharge
  • Itching of the nose and eyes (conjunctivitis)

79
Diagnosis
  • Detailed allergy history (do the symptoms present
    year-round or only during contact with certain
    animals or plants).
  • Seasonal allergic rhinitis, a bluish-purple
    discoloration of the mucosa.
  • Perennial rhinitis, the mucosa is bright red and
    shows inflammatory changes.
  • Careful allergy testing is necessary to identify
    the antigens involved.

80
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81
Treatment
  • The best treatment strategy is to avoid contact
    with the allergen or eliminate allergenic
    irritants
  • Pharmacologic treatment
  • Mast-cell stabilizers
  • Local and systemic H1 antihistamines
  • Local steroids
  • Immunotherapy or hyposensitization therapy
  • Surgical options

82
Vasomotor Rhinitis
  • Resembles allergic rhinitis in its clinical
    features, but there is no evidence that the
    patient has been previously sensitized.
  • Neurovascular autonomic disturbances in
    regulating the tonus of the nasal mucosal vessels

83
Symptoms
  • Obstructed nasal breathing
  • Watery nasal discharge
  • Sneezing
  • The history shows that the symptoms are related
    to a temperature change, the consumption of hot
    liquid or alcohol, or less specifically to
    emotional stress.

84
Treatment
  • Medical therapy includes
  • Antihistamines
  • corticosteroid-containing nasal sprays
  • In the Kneipp system of therapy, ice-cold water
    is sniffed up the nose as a way of training the
    neuroautonomic regulation of the blood supply to
    the nasal mucosa

85
Traetment
  • For intractable vasomotor rhinitis is surgical
    reduction of the turbinates a septoplasty should
    be performed.

86
Atrophic Rhinitis
  • Characterized by pronounced dryness of the nasal
    mucosa.
  • Severe cases, especially with secondary bacterial
    colonization, are marked by a fetid nasal odor
    that is not perceived by the patient due to
    degeneration of the olfactory epithelium.

87
Etiology
  • Primary atrophic rhinitis is unknown
  • Secondary forms
  • Extensive prior tumor resection
  • Excessive use of nose drops drug abuse (cocaine)
  • Previous radiotherapy for nasal and sinus tumors

88
Treatment
  • Conservative
  • Symptomatic measures (saline nasal douche,
    soothing mucosal ointments).
  • Surgery
  • reduce the nasal cavity by the submucous
    implantation of cartilage grafts.

89
Hormonal Rhinitis
  • Occurs mainly during pregnancy and is believed to
    be caused by estrogen-induced swelling of the
    mucosa with nasal airway obstruction.

90
Rhinitis Medicamentosa
  • This disease occurs mainly as a side effect from
    the long-term use of decongestant nose drops

91
Drug induced Rhinitis
  • Antihypertensive drugs
  • Beta-blockers,
  • Angiotensin-converting enzyme (ACE) inhibitors
  • Oral contraceptive
  • Clinical
  • symptoms consist of obstructed nasal
    breathing, dry mucosa, and occasional olfactory
    disturbances.

92
Chronic Sinusitis
  • Intranasal anatomic changes such as
  • Septal deviation
  • Septal spurs
  • Chronic inflammation
  • Allergy
  • Trauma
  • Neoplasms
  • The common pathogenic mechanism is impaired
    ventilation of the ostiomeatal unit

93
  • Chronic sinusitis frequently affects the
    maxillary sinus and ethmoid cells

94
Symptoms
  • Pain (from feeling of pressure to persistent or
    recurrent headaches)
  • Nasopharyngeal drainage (postnasal drip)
  • Obstructed nasal breathing

95
Diagnosis
  • Rhinoscopy
  • Endoscopy
  • Imaging studies

96
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97
Treatment
  • Conservative treatment options
  • Appropriate antiallergic therapy
  • Sinus surgery

98
Endoscopic sinus surgery
  • The modern surgical treatment of chronic
    sinusitis is performed intranasally under
    endoscopic or microscopic control.

99
Nasal Polyposis
  • Genetic causes
  • Chronic irritation of the mucosa, like that
    occurring in chronic rhinitis or sinusitis
  • In response to allergic rhinitis and
    acetylsalicylic acid (ASA) intolerance

100
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101
Notice
  • Nasal polyps are rarely observed in children.
  • Most occur in a setting of cystic fibrosis.

102
Symptoms
  • Obstructed nasal breathing
  • Hyposmia or anosmia
  • Headache
  • Snoring
  • Rhinophonia clausa
  • Frequent throat clearing
  • Spread to the lower airways can lead to
    laryngitis with hoarseness and bronchitic
    symptoms.

103
Diagnosis
  • Rhinoscopic or endoscopic evaluation
  • Computed tomography
  • Allergy tests
  • Olfactory testing

104
Treatment
  • Conservative measures
  • Use of corticoid containing nasal sprays
  • Systemic antihistamines
  • Systemic steroids
  • Surgical treatment

105
Prognosis
  • The prognosis is guarded even with modern
    surgical techniques most meticulous ablative
    sinus surgery cannot prevent a recurrence

106
Mucoceles and Pyoceles
  • Adhesions due to
  • Postinflammatory
  • Post-traumatic
  • Postoperative
  • The most common site of occurrence is the frontal
    sinus, followed by the ethmoid cells, maxillary
    sinus, and sphenoid sinus.

107
Frontal Sinus
  • Presents as an isolated, tense swelling over the
    anterior wall of the frontal sinus
  • It may also cause inferolateral displacement of
    the orbital contents

108
Maxilla
  • Swelling in the cheek area with upward
    displacement of the orbital contents

109
Ethmoid
  • Proptosis, limited ocular movements, and diplopia
    may also occur, depending on the location of the
    mass.

110
Diagnosis
  • Computed tomography
  • MRI

111
Treatment
  • The treatment of choice is surgical removal of
    the mucocele

112
Rhinosinogenic Complications
113
Orbital Complications
  • They occur with highest frequency in children
    under 6 years of age
  • Orbital edema
  • Periosteitis
  • Subperiosteal abscess
  • Orbital cellulitis
  • Orbital apex syndrome
  • Cavernous sinus thrombosis

114
Orbital Cellulitis
115
Bone and Soft-Tissue Inflammations
116
Osteitis and Osteomyelitis
  • Osteomyelitis occurs mainly as a complication of
    frontal sinusitis

117
Symptoms
  • The patient presents clinically with a tender,
    doughy, erythematous swelling over the forehead

118
Diagnosis
  • Cranial CT scans

119
Treatment
  • The treatment of choice is surgical eradication
    of the affected bone under antibiotic coverage

120
Intracranial Complications
  • Epidural, subdural and intracerebral abscesses
  • Meningitis
  • Sinus Thrombosis and Thrombophlebitis

121
Question
  • What is so serious regarding nasal foliculitis?
  • Name the common symptoms of sinusitis.
  • When orbit shift to the inferolateral the mucocel
    perhaps is located in . sinus.
  • Name the causes of sinonasal polyposis.
  • Subdural abscess is more common when the ..
    Sinus is involved.
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