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Bell

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Bell s Palsy Bell s Palsy Characterized by: Peripheral facial paralysis Acute benign cranial polyneuritis Acute disorder characterized by a disruption of the ... – PowerPoint PPT presentation

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Title: Bell


1
Bells Palsy

2
Bells Palsy
  • Characterized by
  • Peripheral facial paralysis
  • Acute benign cranial polyneuritis
  • Acute disorder characterized by a disruption of
    the motor branches of cranial nerve VII on one
    side of the face. (in absence of stroke)

3
Bells Palsy
  • Can affect any age group, though more common from
    20-60.
  • Etiology unknown though reactivated herpes
    simplex may be involved.
  • Reactivation causes edema, inflammation,
    ischemia, and eventual demyelination of the
    nerve, creating pain and alteration in motor and
    sensory function.

4
Clinical manifestations
  • Benign, with 85 of people recovering in 6
    months-remaining 15 have some asymmetry of
    facial muscles

5
Clinical manifestations
  • Often accompanied by an outbreak of herpes
    vesicles in or around the ear.
  • Pain around or behind the ear
  • Fever, tinnitus, hearing deficits
  • Flaccidity of the affected side of the face with
    drooping of the mouth accompanied by drooling DT
    paralysis of the facial nerve (motor branches)

6
Clinical manifestations
  • Inability to close the eyelids, with an upward
    movement of the eyeball when closure is
    attempted lower lid may turn out
  • Wide palpebral fissure (opening between eyelids)
  • Flattening of the nasolabial fold
  • Inability to smile, frown, or whistle
  • Unilateral loss of taste
  • Altered chewing ability loss of or excessive
    tearing

7
Complications
  • Psychological withdrawal DT changes in
    appearance,malnutrition or dehydration, mucous
    membrane trauma, corneal abrasion, muscle
    stretching, and facial spasms and contractures.

8
Diagnostic Studies
  • Diagnosis made on basis of symptoms in the
    absence of other causes of paralysis such as
    stroke.
  • No definitive test
  • EMG may determine nerve excitability or absence

9
Therapeutic Management
  • Corticosteroids- drug of choice
  • Prednisone may be started immediately!
  • Best if initiated before paralysis is complete
  • Taper off over 2 weeks
  • Decrease edema and pain
  • Analgesics may be needed for pain
  • Antivirals Acyclovir (Zovirax) and Famvir
    because HSV is implicated in 70 of cases.
  • See Lewis 1719-1720- Nursing Implementation
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