FLUID AND ELECTROLYTE BALANCES Ms. Ida Anitha Lecturer College of Nursing CMC, Vellore - PowerPoint PPT Presentation

1 / 44
About This Presentation
Title:

FLUID AND ELECTROLYTE BALANCES Ms. Ida Anitha Lecturer College of Nursing CMC, Vellore

Description:

Water is found everywhere on earth including human body. In an adult 60% of the ... Finger print impression on the sternum after palpation. Personality change ... – PowerPoint PPT presentation

Number of Views:1064
Avg rating:3.0/5.0
Slides: 45
Provided by: Kar7165
Category:

less

Transcript and Presenter's Notes

Title: FLUID AND ELECTROLYTE BALANCES Ms. Ida Anitha Lecturer College of Nursing CMC, Vellore


1
FLUID AND ELECTROLYTE BALANCESMs. Ida
AnithaLecturerCollege of NursingCMC, Vellore
2
WHY IS IT IMPORTANT FOR NURSES TO KNOW ABOUT
FLUID ELECTROLYTE BALANCE
3
INTRODUCTION
  • Water is found everywhere on earth including
    human body
  • In an adult 60 of the weight is water
  • Two third of the bodys water is found in the cell

4
DISTRIBUTION OF BODY FLUIDS
  • Body fluids are distributed in two distinct
    compartments
  • 1.Extracellular fluidsECF Which includes
    interstitial fliud intravascular fluid
  • 2.Intracellular fluidsICF

5
COMPOSITION OF BODY FLUIDS
  • The fluids circulating throughout the body in
    extracellular and intracellular fluid spaces
    contain
  • 1.Electrolytes
  • 2.Minerals
  • 3.Cells

6
MOVEMENT OF BODY FLUIDS
  • Diffusion
  • Osmosis
  • Filtration
  • Active transport

7
REGULATION OF BODY FLUIDS
  • Fluid intake
  • Fluid output
  • Hormonal influence
  • Lymphatic influences
  • Neurologic influences
  • Renal influences

8
ACID-BASE BALANCE
  • Chemical regulation
  • Biologic regulation
  • Physiological regulation
  • 1.Lungs
  • 2.Kidneys

9
FLUID ,ELCTROLYTE AND ACID-BASE IMBALANCES
10
FLIUD IMBALANCES
  • The five types of fluid imbalances that may occur
    are
  • Extracellular fluid imbalances(EVFVD)
  • Extracellular fluid volume excess(ECFVE)
  • Extracellular fluid volume shift
  • Intracellular fluid vloume excess(ICFVE)
  • Intrcellular fluid volume deficit(ICFVD)

11
EXTRACELULLAR FLUID VOLUME DEFICIT
  • An ECFVD, commonly called as dehydration , is a
    decrease in intravascular and interstitial fluids
  • An ECFVD can result in cellular fluid loss if it
    is sudden or severe

12
THREE TYPES OF ECFVD
  • Hyperosmolar fluid volume deficit- water loss is
    greater than the electrolyte loss
  • Isosmolar fluid volume deficit equal proportion
    of fluid and electrolyte loss
  • Hypotonic fluid volume deficit electrolyte loss
    is greater than fluid loss

13
ETIOLOGY AND RISK FACTORS
  • Severe vomiting
  • Diaphoresis
  • Traumatic injuries
  • Third space fluid shifts percardial, pleural,
    pertonial and joint cavities
  • Fever
  • Gatrointestinal suction
  • Ileostomy
  • Fistulas
  • Burns
  • Hyperventilation
  • Decresed ADH secretions
  • Diabetes insipidus
  • Addisons disease or adrenal crisis
  • Diuretic phase of acute renal failure
  • Use of diuretics

14
ELDERLY ARE HIGH RISK OF ECFVD DUE TO
  • Decreased thirst response
  • Decreased renal concentration of urine
  • Altered ADH response
  • Increased drug drug interaction
  • Multiple chronic diseases
  • Decreased access to fluids due to financial or
    transportation barriers
  • Debilitation
  • Chemical or physical restraint
  • Changes in mental status

15
CLINICAL MANIFESTATION
  • In Mild ECFVD, 1to 2 L of water or 2 of the body
    weight is lost
  • In Moderate ECFVD, 3 to 5L of water loss or
    5weight loss
  • IN Severe ECFVD , 5 to 10 L of water loss or 8
    of weight loss

16
CLINICAL MANIFESTATION
  • Peripheral vein fillinggt 5 seconds
  • Postural systolic BP falls gt25mm Hg and
    diastolic fall gt 20 mm Hg , with pulse increases
    gt 30
  • Narrowed pulse pressure, decreased CVPPCWP
  • Flattened neck veins in supine position
  • Weight loss
  • Oliguria(lt 30 mlper hour)
  • Decreased number and moisture in stools
  • Thirst
  • Muscle weakness
  • Dry mucus membranedry cracked lips or furrowed
    tongue
  • Eyeballs soft and sunken (severe deficit)
  • Apprehension , restlessness, headache ,
    confusion, coma in severe deficit
  • Elevated temperature
  • Tachycardia, weak thready pulse

17
LABORATORY FINDINGS
  • Increased osmolality(gt 295 mOsm/ kg)
  • Increased or normal serum sodium level (gt 145mEq/
    L )
  • Increase BUN (gt25 mg / L )
  • Hyperglycemia ( gt120 mg /dl )
  • Elevated hematocrit (gt 55)
  • Increased specific gravity ( gt 1.030)

18
MANAGEMENT
  • Mild fluid volume loss can be corrected with oral
    fluid replacement
  • -if client tolerates solid foods advice to take
    1200 ml to 1500ml of oral fluids
  • -if client takes only fluids, increase the total
    intake to 2500 ml in 24 hours

19
Management of Hyperosmolar fluid volume deficit
  • Administration of hypotonic IV solution , such
    as 5 dextrose in 0.2 saline
  • If the deficit has existed for more than 24
    hours,avoid rapid correction of fluid sodium
    solution to be infused at the rate of 0.5 to 0.1m
    Eq/ L/ hr

20
If heamorrhage is the cause for ECFVD
  • Packed red cells followed by hypotonic IV fluids
    is administered
  • In situations where the blood loss is less than 1
    L normal saline or ringer lactate may be used
  • clients with severe ECFVD accompanied by severe
    heart , liver, or kidney disease cannot tolerate
    large volumes of fluid and sodium

21
EXTRACELLULAR FLUID VOLUME EXCESS
  • ECFVE is increased fluid retention in the
    intravasular and interstitial spaces

22
ETIOLOGY AND RISK FACTORS
  • Heart failure
  • Renal disorders
  • Cirrhosis of liver
  • Increased ingestion of high sodium foods
  • Excessive amount of IV fluids containing sodium
  • Electrolyte free IV fluids
  • SIADH,Sepsis
  • decreased colloid osmotic pressure
  • lymphatic and venous obstruction
  • Cushings syndrome glucocorticoids

23
CLINICAL MANIFESTATION
  • Constant irritating cough
  • Dyspnea crackles in lungs
  • Cyanosis, pleural fffusion
  • Neck vein obstruction
  • Bounding pulse elevated BP
  • S3 gallop
  • Pitting sacral edema
  • Weight gain
  • Increased CVP PCWP
  • Change in level of consiousness

24
LAB INVESTIGATION
  • serum osmolality lt275mOsm/ kg
  • Low , normal or high sodium
  • Decreased hematocrit lt 45
  • Specific gravity below 1.010
  • Decreased BUN lt 8mg/ dl

25
MANAGEMENT
  • Diuretics combination of potassium sparing and
    potassium depleting diuretics
  • In people with CHF, ACE inhibitors and low dose
    of beta blockers are used
  • A low sodium diet

26
EXTRACELLULAR FLUID VOLUME SHIFT THIRD SPACING
  • Fluid that shifts into the interstitial spaces
    and remain there is called as third space fluid
  • Common sites are abdomen , pleural cavity,
    peritoneal cavity and pericardial sac

27
RISK FACTORS
  • Crushing injuries, major tissue trauma
  • Major surgery
  • Extensive burns
  • Acid base imbalances and sepsis
  • Perforated peptic ulcers
  • Intestinal obstruction
  • Lymphatic obstruction
  • Autoimmune disorders
  • Hypoalbunemia
  • GI tract malabsorption

28
CLINICAL MANIFESTATION
  • skin pallor
  • Cold extremities
  • Weak and rapid pulse
  • Hypotension
  • Oliguria
  • Decreased levels of consiousness
  • LAB INVESTIGATION
  • Elevated hematocrit BUN level

29
MANAGEMENT
  • Treat the cause
  • For burns and tissue injuries large volume of
    isosmolar IV fluid is administered
  • Albumin is administered for protein deficit
  • IV fluid intake is maintained after major
    surgery to maintain kidney perfusion
  • Pericardiocentesis if pericarditis is the result
  • Paracentesis for ascitis

30
INTRACELLULAR FLUID VOULME EXCESSWATER
INTOXICATION
  • ICFVE is increase in amount of water inside the
    cells

31
ETIOLOGY
  • Administration of excessive amount of hyposmolar
    IV fluids0.45saline or 5dextrose in water
  • Consumption of excessive amount of tap water
    without adequate nutritional intake
  • SIADH
  • Schizophreniacompulsive water consumption

32
CLINICAL MANIFESTATIONS
  • Headaches
  • Behavioral changes
  • Apprehension
  • Irritability, disorientation and confusion
  • Increased ICP pupillary changes and decreased
    motor and sensory function
  • Bradycardia, elevated BP, widened pulse pressure
    altered respiratory patterns, Babinskis
    response flaccidity, projectile vomiting,
    Papilledema, delirium, convulsions coma

33
LABORATORY FINDINGS
  • High serum sodium level- 125 mEq/L
  • decreased hamatocrit

34
MANAGEMENT
  • Early administration of IV fluids containing
    sodium chloride cam prevent SIADH
  • oral fluids such as juices or soft drinks can be
    given orally every hour
  • Perform neurologic checks every hour to see if
    cranial changes are present
  • Monitor fluid intake , IV fluids and fluid output
    hourly and weight daily
  • Administer antiemetics for food and fluid
    retention

35
INTRACELLULAR FLUID VOLUME DEFICIT
  • Severe hypernatremia and dehydration can cause
    ICFVD
  • Relatively rare in healthy adults
  • common in elderly people and in those conditions
    that result in acute water loss
  • Symptoms include confusion, coma, and cerebral
    hemorrhage

36
 
37
 
38
(No Transcript)
39
 
40
 
41
 
42
 
43
 
44
CONCLUSION

Thank you
Write a Comment
User Comments (0)
About PowerShow.com