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Cardiovascular Assessment

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Place stationary finger in person's fifth ... Using 1 finger locate the apical impulse ... palpate carotid pulse with index & middle finger as listen to heart sounds ... – PowerPoint PPT presentation

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Title: Cardiovascular Assessment


1
Cardiovascular Assessment
  • The Heart
  • Extends from the 2ed to the 5th intercostal
    space
  • Between the R boarder of the sternum to the L
    midclavicular
  • Beats against chest wall to produce apical
    impulse
  • Palpate 5th intercostal space 7-9cm form the mid
    sternal line

2
  • Heart Chambers
  • Right atrium (RA)
  • Right ventricle (RV)
  • Left atrium (LA)
  • Left ventricle (LV)
  • Valves
  • Aortic valve
  • Mitral valve
  • Tricuspid valve
  • Pulmonic valve

3
  • Directions of blood flow
  • Blood Flow
  • From liver to right atrium (RA) via inferior vena
    cava Superior vena cava drains venous blood from
    the head and upper extremities
  • From RV, venous blood travels through tricuspid
    valve to right ventricle (RV)
  • From RV, venous blood flows through pulmonic
    valve to pulmonary artery
  • Pulmonary artery delivers unoxygenated
  • blood to lungs
  • 3.Lungs oxygenate blood

4
  • Pulmonary veins return fresh blood to LA
  • 4. From LA, arterial blood travels through mitral
    valve to LV. LV ejects blood through aortic
    valve into aorta
  • Aortic delivers oxygenated blood to body
  • Diastole
  • Ventricles relax and fill with blood and AV
    valves (tricuspid, mitral) open
  • Bottom number of the B/P
  • Occurs when AV valves are open to allow filling
    of the ventricles

5
  • Systole
  • Heart contract and blood pumped in ventricles and
    fills pulmonary systemic arteries
  • Closure of AV valves contributes to first heart
    sound and signals beginning of systole
  • Top number of B/P

6
  • Percussion outline of the hearts boarder
    limited with the female breast tissue or in an
    obese person or person with muscular chest wall.
  • Place stationary finger in persons fifth
  • intercostal space over on L side of chest near
    anterior axillary line. Slide hand toward
    yourself note change in sound from resonance over
    the lung to dull( over the heart)
  • Left border of cardiac dullness is at the
    midclavicular line in 5th interspace

7
  • and slopes toward sternum
  • At the second interspace the border os dullness
    coincides with left sternal border
  • Right border of dullness matches the sternal
    border
  • Palpate apical impulse
  • Using 1 finger locate the apical impulse
  • Ask client to exhale and holdaids in locating
    pulsation
  • May need to turn client to left to find it.
  • Note Location, size, amplitude and Duration

8
  • Apical impulse palpable in ½ adults, and not in
    obese or thick wall clients.
  • Apical impulse increases in amplitude and
    duration in client with anxiety, fever,
    hyperthyroidism and anemia
  • Auscultation listen for the pitch, rate,
    regularity,
  • -low pitched, short duration of sound,
    regularity
  • -deep breathing will temporarily slow heart rate

9
  • -identify S1 and S 2, assess S 1and S 2
    separately, listen for extra heat sounds and
    listen for murmurs
  • -with stethoscope use Z pattern from base
    of the heart across and down, then over apex
    or start at apex and work way up.
  • -note rate and rhythm
  • -if notice irregularity, check for pulse
    deficit
  • (check radial and apical pulse
    simultanously)

10
  • When listening
  • Start at base of heart and use Z pattern.
  • Note
  • -rate rhythm
  • -Identify S1 and S2
  • -Assess S1and S2 separately
  • -Listen for extra heart sounds
  • -Listen for murmurs
  • Landmarks
  • -second R interspace aortic valve area

11
  • -second L interspace pulmonic valve area
  • -L lower sternal border triscupid valve area
  • -fifth interspace at around L midclavicular
    line mitral valve area
  • Heart Sounds
  • S1
  • Closure of the AV valve
  • Beginning of systole
  • lup of the lup-dup sound
  • -louder at apex
  • -coincides with carotid artery pulse

12
  • S2
  • closure of semilunar valves
  • end of systole
  • dup of the lup-dup sound
  • louder at base
  • Abnormal Heart Sounds
  • Split S
  • -benign condition occurring in some patients
    upon inspiration
  • -lup-t-dup sound
  • -crisper than a murmur
  • -occurs at end of inspiration
  • -occurs about every fourth beat, fading in
    with
  • inhalation and out with exhalation

13
  • Murmurs
  • Vibrations within the hearts chambers or major
    arteries from the back and forth blood flow
    swishing sound
  • Innocent- no anatomic or physiologic abnormality
    exists
  • Functional- no anatomic cardiac defect exists but
    physiologic abnormality such as anemia, fever,
    pregnancy, hyperthyroidism)

14
  • Murmur
  • Blowing, swooshing sound that occurs with
    turbulent blood flow in the heart or great
    vessels.
  • Need to ID if it occurs in systole or diastole
  • Extra humming sound between S1 and S2 sounds
  • Listen to pitch (high, medium, or low)
  • Listen to pattern (crescendo, decrescendo,
  • crescendo-decrescendo or diamond shaped)
  • -Listen to loudness (grades i to vi barely
    audible to loudness)

15
  • -Quality (musical, blowing, harsh, rumbling)
  • -Location ( where best heard)
  • -Radiation-transmitted downstream direction of
    blood flow
  • -Posture- disappear or enhanced by change of
    position
  • Causes
  • structural- weakened valve or wall defect
    secondary to streptococcal infection or
    congenital defect
  • flow murmur
  • change in blood viscosity secondary to anemia
  • change in blood velocity secondary to exercise

16
  • -structural- weakened valve or wall defect
    secondary to streptococcal infection or
    congenital defect
  • -flow murmur
  • change in blood viscosity secondary to anemia
  • change in blood velocity secondary to exercise

17
  • Loudness
  • --Grades i-vi(adults)
  • i barely audible, quiet room
  • ii clearly audible but faint
  • iii moderately loud
  • ivloud, associated with a thrill palpable on
    chest wall
  • v very loud, heard with one corner of
  • steth. lifter off chest wall
  • viloudest, still heard with entire steth.
    lifted
  • just off the chest wall

18
  • Thrill
  • Palpable vibration felt over the heart as blood
    moves from chamber to chamber
  • ALWAYS ABNORMAL
  • Pericardial Friction Rub
  • Grating sound upon inspiration, stops when breath
    is held
  • ALWAYS ABNORMAL

19
  • Assessment of Peripheral Pulses Strength
  • 0absent
  • 1weak
  • 2diminished
  • 3strong
  • 4bounding and full
  • Assessment of Pulses
  • Regularity/ Rate
  • Bilateral Equality
  • Should be the same on both sides of the body L or
    R

20
  • Head to Toe Equality
  • Should not be greatly different head to toe
  • Use additional assessments
  • Check against apical pulse
  • Check other measures of circulation if pulse
    heard to find

21
Laeral to
  • Cardiac Assessment in Children
  • Inspection notice color of skin and mucous
    membranes, observe child in semi-fowler position,

  • check for edema, warmth of extremities
  • Palpation locate apical impulse (AI)
  • -Lateral to the L midclavicular (LMCL) and
    fourth ICS in children
  • -At LMCL and fifth ICS in children 7 yrs. old
  • Point of maximum intensity (PMI)- area of most
    intense pulsation Ai and PMI not used
    interchangeably but they are at the same place
  • Thrills- palpable vibrations best felt with

-
22
  • ball of hand and during expiration
  • produced by flow of blood from one
  • chamber of heart to another through
  • narrow or abnormal opening
  • Pericardial friction rubs-scratchy, high pitched
    grating sound not affected by changes in
    respirations
  • Capillary refill time-brisk-less than 2 seconds
    blanch nail beds with pressure for a few seconds
    and then release prolonged associated with poor
    systemic perfusion

23
  • Auscultation (Children)
  • S1 and S2 correspond to lub-dub
  • S1 caused by closure of tricuspid and mitral
    valves
  • S2 caused by closure of pulmonic and aortic
    valves
  • Distinguish between S1 and S2 simultaneously
    palpate carotid pulse with index middle finger
    as listen to heart sounds

24
  • Alterations Through the Lifespan (Children)
  • Infants and Children
  • Change from fetal to neonatal circulation in
    first 24 hours
  • Foramen ovale closes in1st hours
  • ductus arterious closes by the first weeks of
    life
  • Heart is placed more laterally in children
  • Apex is at 4th intercostal space before 3 yrs
  • Apex is at the 5th intercostal space at 7 yrs.
    age
  • CAROTID ARTERY IN NOT PALPATED IN PATIENTS UNDER
    ONE YEAR OF AGE

25
  • Abnormal findings in Pediatric patients
  • Murmurs common and usually outgrow
  • Remain aware of strep effects on the heart
  • valve
  • Innocent (no valvular or pathologic cause it
    is just a noise
  • Functional (due to increase blood flow) need to
    have diagnosis test as EKG or echocardiogram
  • Rheumatic fever-
  • Causes weakening of heart valve

26
  • Physiological Changes in Pregnancy
  • Growing uterus pushes heart up. Left and forward
  • Blood volume rises to 40-45 greater than pre
    pregnancy vol.
  • Murmur in 90 pregnant patients disappear at
    delivery ( non-functional murmurs)
  • Cardiac output rises to 50 above pre pregnancy
    levels pulse rate rises by 10-20
  • In Vessels
  • Vena Cava syndrome
  • Uterus puts pressure on vena cava when patients
    is in supine position

27
  • Lowers B/P
  • Patient feels clammy, dizzy and shows pallor
  • Turn patient to L side to relieve pressure of
    vena cava

28
  • Geriatrics
  • Arteries stiffen with age and B/P rises
  • Average rise is 20 mmHg between 20-60 and between
    60-80 another 20 mm HG
  • LV wall thickens
  • Loss of ability to augment exercise with
    increased cardiac output
  • Resting pulse rate remain in the lifetime range
    between 60-100 and maybe irregular
  • Vessels lose elasticity
  • Varicose veins
  • More susceptible to clots

29
  • Loss of venous elasticity plus less efficient
    cardiac output results in decreased circulation
  • Lower healing
  • Body temperature, regulation is changed
  • Abnormal findings in Geriatric Patients
  • At risk for dehydration and altered nutritional
    status
  • Dehydration impacts fluid volume and B/P
  • Electrolyte imbalance impact on
    cardioelectricity

30
  • Murmurs common in this age group
  • Arryhthmias more common in this age group
  • Ectopic beats( extra beats)
  • Lower cardiac output and B/P
  • May deprive organs of needed oxygenation
  • Better tolerated in younger population
  • Tachycardia
  • Results in 40-70 drop in cerebral blood volume
  • Syncope

31
  • Peripheral blood vessels grow more rigid with
    age
  • Tests and vocabulary
  • Atherosclerosis deposit of fatty plaques on the
    intima of the arteries.
  • Artheriosclerosis arteries becoming more rigid
    producing a rise in systolic B/P
  • Claudication- pain produced when walking, not
    relieved by rest
  • Bruit- occurs with turbulent blood flow,
    indicating partial occlusion

32
  • Modified Allen Test- evaluate collateral
    circulation firmly depress ulna radial arties,
    patient opens closes fist normal when open
    fist, blood returns to normal
  • Homans sign-pain in calf with dorsiflexion of
    the foot, indicating thrombophlebitis or
    thrombus
  • Pitting Edema
  • 1 mild pitting, slightly indentation, no
    perceptible swelling of legs depth of pitting is
    1 cm
  • 2 moderate pitting, indentation subsides
    rapidly depth of pitting is 2 cm

33
  • 3 deep pitting, indentation remains for a short
    time, leg looks swollen depth of pitting is 3
    cm
  • 4 very deep pitting, indentation lasts a long
    time, leg is very swollen depth of pitting is 4
    cm
  • Trendelenburg Test- varicosites present in legs
    to determine valve competence lying supine
    elevate legs 90 until veins empty, place
    tourniquet high on thigh, help patient to
  • Stand, watch for venous filling saphenous veins
    should fill slowly from below in about 30 seconds

34
  • Taking a Health History
  • Lifestyle factors
  • Smoking- stimulant for CV system
  • Serum cholesterol- causes blockages
  • Obesity- stresses heart with fat deposits and
    constant state of exertion
  • Past medical history
  • Diabetes- stresses body, effects heart
  • HTN- wear tear in aorta and LV
  • Family history heart disease or PVD
  • Large genetic correlation

35
  • Problems during pregnancy
  • Are children meeting developmental milestones
    expected growth parameters
  • Any history of chest pain
  • When
  • What precipitated it
  • What gave relief
  • Qualities
  • Stabbing, crushing, shooting, radiating
  • Angina
  • Constriction of small vessels surrounding the
    heart
  • Causes sharp chest pain

36
  • Relief with nitroglycerine
  • Assess for complains of fatigue, pallor, edema
    and temperature alterations in the extremities
  • All indicate possible poor cardiac output
  • Nursing Interventions
  • Take a careful history
  • Provide patient teaching as to risk factors for
    cardiovascular health
  • When helping patients OOB allow them to move

37
  • to prevent hypotension
  • Encourage pregnant women to lay on their L side
    to prevent vena cava syndrome
  • Educate pregnant women of S S of pre eclampsia
    and keep careful B/P records of patients
  • Keep careful records of pediatric growth
    development parameters for each patient and
    continually check progress
  • Watch oxygen status of patient with altered
    cardiac output
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