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Morning Report 73107

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Target lesions (Lyme) Splinter hemm, Osler nodes, etc (endocarditis) ... Lyme disease, endocarditis, Rheumatic fever, Chagas disease, myocarditis. Rheumatic disease ... – PowerPoint PPT presentation

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Title: Morning Report 73107


1
Morning Report7/31/07
  • 3rd Degree AV block
  • Jason Haag

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Heart Block
  • 1st Degree AV Block
  • one-to-one relationship exists between P waves
    and QRS complexes, but the PR interval is longer
    than 200 ms

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Heart Block
  • 2nd Degree Mobitz Type I AV Block (Wenckebach)
  • PR interval is prolonging with each P wave to the
    point when the P wave is no longer conducted

6
Heart Block
  • 2nd Degree Mobitz Type II AV Block
  • PR interval is constant, but occasionally P waves
    are not followed by the QRS complexes

7
Heart Block
  • 3rd Degree Heart Block
  • More P waves than the QRS complexes exist and no
    relationship exists between them

8
3rd Degree Heart Block
  • Block can be in AV node or infranodal conduction
    system
  • AV node
  • 2/3 escape rhythms have narrow QRS (junctional)
  • Fascicular or bundle branches
  • Wide QRS (subjunctional)
  • Rate typically in low 40s

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Frequency
  • In the US 0.02
  • Internationally 0.04.
  • Age Bimodal peak, at infancy given congenital
    complete AV block and at advance d age due to
    progressive fibrosis and ischemia

10
History
  • Syncope, near-syncope, and lightheadedness
  • Fatigue, dyspnea, and angina
  • Asymptomatic
  • Sudden cardiac death

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Physical
  • Vital Signs (stable vs. unstable, always check HR
    manually)
  • Signs of heart failure JVD, a waves, Pulmonary
    edema
  • New murmurs or gallops
  • Target lesions (Lyme)
  • Splinter hemm, Osler nodes, etc (endocarditis)
  • Neuromuscular changes (mytonic/muscular
    dystrophy)

12
Etiologies
  • Idiopathic Progressive Cardiac Conduction Disease
  • ½ of cases of AV block
  • Lenegres disease
  • Progressive, fibrotic, sclerodegeneration of the
    conduction system
  • Younger individuals, may be hereditary
  • Levs disease
  • Calcification extending from fibrous structures
    (aortic/mitral rings) into the conduction system
  • Older individuals, ? ESRD
  • Fibrosis NOS
  • Typically mitral and aortic rings
  • Mitral ? narrow QRS
  • Aortic ? wide QRS

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Etiologies (cont.)
  • Ischemic heart disease
  • 40 of cases
  • Either from chronic ischemia or acute MI
  • Acute MI AV blocks (20 of patients)
  • 1st degree (8)
  • 2nd degree (5)
  • 3rd degree (6)
  • LBBB/RBBB (10-20)
  • AV nodal block (narrow QRS) associated with
    inferior wall MI
  • Bundle blocks (wide QRS) associated with anterior
    wall MI
  • Drugs
  • Calcium channel blockers, beta blockers, digoxin,
    amiodarone, adenosine, quinidine, procainamide

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Etiologies (cont.)
  • Infection
  • Lyme disease, endocarditis, Rheumatic fever,
    Chagas disease, myocarditis
  • Rheumatic disease
  • Ankylosing spondylitis, Reiter syndrome,
    relapsing polychondritis, rheumatoid arthritis,
    scleroderma
  • Infiltrative disease
  • Amyloidosis, sarcoidosis, multiple myeloma,
    hemachromatosis, Wilsons disease

15
Etiologies
  • Hyperthyroidism
  • Metabolic
  • Hypoxia, hyperkalemia
  • Neuromuscular disease
  • Muscular dystrophy, dermatomyositis

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Treatment
  • Correct underlying problem if you can
  • Correct K, stop AV blocking medications, etc.
  • If unstable
  • Transcutaneous pacing
  • If stable
  • Plan for permanent pacemaker placement

17
Permanent Pacemaker
  • Class I - Conditions for which evidence and/or
    general agreement exists that a given procedure
    or treatment is beneficial, useful, and effective
  • Third-degree AV block and advanced second-degree
    AV block at any anatomic level associated with
    any one of the following conditions
  • Bradycardia with symptoms, heart failure,
    arrhythmias, pauses greater than 3 seconds,
    escape rate

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Permanent Pacemaker
  • Class IIa - Weight of evidence or opinion is in
    favor of usefulness or efficacy
  • Asymptomatic third-degree AV block at any
    anatomic site with average awake ventricular
    rates of 40 bpm or faster, especially if
    cardiomegaly or left ventricular (LV) dysfunction
    is present

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References
  • Gregoratos G, Abrams J, Epstein AE, et al
    ACC/AHA/NASPE 2002 guideline update for
    implantation of cardiac pacemakers and
    antiarrhythmia devices summary article a report
    of the American College of Cardiology/American
    Heart Association Task Force on Practice
    Guidelines. Circulation 2002 Oct 15 106(16)
    2145-61.
  • Kojic EM, Hardarson T, Sigfusson N, Sigvaldason
    H The prevalence and prognosis of third-degree
    atrioventricular conduction block the Reykjavik
    study. J Intern Med 1999 Jul 246(1) 81-6.
  • McEnvoy GK, ed AHFS Drug Information 2000.
    Bethesda, Md American Society of Health-System
    Pharmacists 2000 1187-95.
  • Ostaner LD, Brandt RL, Kjelsberg MI, et al
    Electrocardiographic findings among the adult
    population of a total natural community. 1965
    31 888-98.
  • Rardon DA, Miles WM, Mitrani RD, et al
    Electrocardiographic Recognition
    Atrioventricular Block and Dissociation. In
    Zipes DP, Jalife J, eds. Cardiac
    Electrophysiology From Cell to Bedside, 2nd ed.
    Philadelphia, Pa WB Saunders 1995.
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